Background:
Macrolides are used for Streptoccoccus pyogenes (Group A Streptococcus, GAS) infections treatment in case of β-lactam allergy. Macrolide resistance (MR) is mainly mediated by methylases [erm(B) and erm(TR)] and efflux pumps [mef(A)]. These genes located into different genetic elements, mainly transposons of the Tn916-family.
Methods:
All GAS clinical isolates collected from adults from 2019 to 2020 were included. The antibiotic susceptibility was studied by microdilution and disk-diffusion methods. MR isolates were typed by emm-typing and pulsed-field gel electrophoresis (PFGE). MR-genes [mef(A), erm(B), and erm(TR)] were detected by PCR. Putative mobile genetic elements (MGE) were determined using linkage experiments and PCR detection of Tn916-family related genes.
Results:
The overall MR rate was 13.6% (35/267), from these MR GAS 20 (57.14%) had cMLSB phenotype (macrolide, lincosamides and streptogramins group B resistant, constitutively expressed), 9 (25.71%) iMLSB (expression induced) and 6 (17.14%) with an M (only macrolide-resistance) phenotype. Seventeen different emm-types were identified, among which the most frequent were emm11 (n=5), emm77 (n=4), emm81 (n=4), emm75 (n=3), emm94 (n=3), emm169 (n=3), emm9 (n=2) and emm219 (n=2) which accounted for 74.29% of all MR-GAS isolates. MR-GAS emm11 isolates harboured erm(B) encoded into transposons of Tn916-family, emm77 was only associated to erm(TR) and tet(O) and emm81 isolates contained mef(A/E).
Conclusions:
Macrolide resistance rates remained stable between 2019-2020 regarding previous periods in our area. Macrolide-resistance determinants were associated to both classical transposons but also, we have obtained results suggesting the spread of no previously described elements.